Loading
Life sciences · Journal article
National Journal of Medical Research · October 1, 2026
No summary has been generated for this record yet. What follows is drawn from its source metadata only.
Journal article.
No findings were extractable from the material analysed.
Safety was not reported in the material analysed. Check the source before drawing any conclusion about harm.
The source did not state who this applies to in practice.
Graded across the dimensions that decide whether you should act, each from what the source actually supports. There is no single score, and where a dimension was not assessed it says so.
This record has not been graded across any dimension yet. Treat the label above as provisional and read the source.
What is missing. This record has no bottom line, key findings, reported figures, evidence dimensions. That is a gap in the analysis, not a judgement about the study.
Background: Diabetic myopathy is an under-recognized complication of Type 2 Diabetes Mellitus (T2DM) that significantly contributes to physical disability. The objective was to estimate the frequency and clinical characteristics of myopathy in T2DM patients using clinical evaluation and electromyography (EMG). Methods: This cross-sectional study evaluated 600 patients with T2DM (duration >5 years) at a tertiary care center. Comprehensive assessments included clinical history, anthropometric measurements, biochemical profiles, and EMG. Myopathy was diagnosed based on clinical features and confirmed through neurophysiological findings. Diabetic myopathy was diagnosed based on clinically demonstrable muscle weakness confirmed by EMG evidence of a myopathic pattern after exclusion of alternative neuromuscular disorders. Results: Myopathy was identified in 85 patients, yielding a frequency of 14.2%. The condition peaked in the 61-70 age group (41.2%) and was more prevalent in males (57.6%). Key correlates included obesity (52.9%) and prolonged disease duration (>15 years). Muscle weakness was a universal finding (100%), with a marked predominance of proximal involvement (61.2%) over distal patterns. Notably, the condition was largely painless (94.1%) with an absence of muscle tenderness. Biochemical analysis revealed poor glycemic control (mean HbA1c 10.33%) and near-normal CK-NAC levels (mean 90.04 U/L), reflecting a non-inflammatory metabolic etiology. Conclusion: Diabetic myopathy affects a significant proportion of patients with long-standing T2DM. It typically presents as a painless, proximal weakness driven by chronic metabolic derangement. Incorporating musculoskeletal screening into routine diabetic management is essential for early detection and the implementation of interventions to preserve motor function and independence.