AUG 17, 2026 · JOURNAL ARTICLE
A Regulatory Element in the Intrinsically Disordered C-Terminal Region of LMTK3 Modulates Its Kinase Domain Interactions and Breast Cancer Phenotypes
Cells
This is a mechanistic study using computational modelling, biophysical assays, and cell/animal models to characterize a regulatory phosphorylation site; it raises a mechanism of LMTK3 regulation rather than testing a clinical hypothesis in patients.
Reported
C-terminal region 1 interacting r…688–1095
C-terminal region 2 interacting r…1181–1486
α-helix 2 residues1247–1258